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HIV latency and integration site placement in five cell-based models

Overview of attention for article published in Retrovirology, August 2013
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Title
HIV latency and integration site placement in five cell-based models
Published in
Retrovirology, August 2013
DOI 10.1186/1742-4690-10-90
Pubmed ID
Authors

Scott Sherrill-Mix, Mary K Lewinski, Marylinda Famiglietti, Alberto Bosque, Nirav Malani, Karen E Ocwieja, Charles C Berry, David Looney, Liang Shan, Luis M Agosto, Matthew J Pace, Robert F Siliciano, Una O’Doherty, John Guatelli, Vicente Planelles, Frederic D Bushman

Abstract

HIV infection can be treated effectively with antiretroviral agents, but the persistence of a latent reservoir of integrated proviruses prevents eradication of HIV from infected individuals. The chromosomal environment of integrated proviruses has been proposed to influence HIV latency, but the determinants of transcriptional repression have not been fully clarified, and it is unclear whether the same molecular mechanisms drive latency in different cell culture models.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 156 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
United States 2 1%
United Kingdom 1 <1%
Denmark 1 <1%
Germany 1 <1%
Unknown 151 97%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 39 25%
Researcher 35 22%
Student > Bachelor 17 11%
Student > Master 14 9%
Professor 9 6%
Other 18 12%
Unknown 24 15%
Readers by discipline Count As %
Agricultural and Biological Sciences 48 31%
Biochemistry, Genetics and Molecular Biology 33 21%
Medicine and Dentistry 23 15%
Immunology and Microbiology 14 9%
Engineering 3 2%
Other 9 6%
Unknown 26 17%