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Mendeley readers
Title |
The progressive ankylosis gene product ANK regulates extracellular ATP levels in primary articular chondrocytes
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Published in |
Arthritis Research & Therapy, October 2013
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DOI | 10.1186/ar4337 |
Pubmed ID | |
Authors |
Ann K Rosenthal, Claudia M Gohr, Elizabeth Mitton-Fitzgerald, Megan K Lutz, George R Dubyak, Lawrence M Ryan |
Abstract |
Extracellular ATP (eATP) is released by articular chondrocytes under physiological and pathological conditions. High eATP levels cause pathologic calcification, damage cartilage, and mediate pain. We recently showed that stable over-expression of the progressive ankylosis gene product, ANK, increased chondrocyte eATP levels, but the mechanisms of this effect remained unexplored. The purpose of this work was to further investigate mechanisms of eATP efflux in primary articular chondrocytes and to better define the role of ANK in this process. |
Mendeley readers
The data shown below were compiled from readership statistics for 35 Mendeley readers of this research output. Click here to see the associated Mendeley record.
Geographical breakdown
Country | Count | As % |
---|---|---|
United Kingdom | 1 | 3% |
Unknown | 34 | 97% |
Demographic breakdown
Readers by professional status | Count | As % |
---|---|---|
Student > Ph. D. Student | 9 | 26% |
Student > Master | 4 | 11% |
Student > Bachelor | 3 | 9% |
Professor | 3 | 9% |
Lecturer | 2 | 6% |
Other | 4 | 11% |
Unknown | 10 | 29% |
Readers by discipline | Count | As % |
---|---|---|
Agricultural and Biological Sciences | 8 | 23% |
Medicine and Dentistry | 4 | 11% |
Biochemistry, Genetics and Molecular Biology | 3 | 9% |
Immunology and Microbiology | 2 | 6% |
Chemistry | 2 | 6% |
Other | 4 | 11% |
Unknown | 12 | 34% |