↓ Skip to main content

Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease

Overview of attention for article published in Molecular Neurodegeneration, December 2013
Altmetric Badge

About this Attention Score

  • Average Attention Score compared to outputs of the same age
  • Above-average Attention Score compared to outputs of the same age and source (57th percentile)

Mentioned by

twitter
3 tweeters

Citations

dimensions_citation
72 Dimensions

Readers on

mendeley
100 Mendeley
You are seeing a free-to-access but limited selection of the activity Altmetric has collected about this research output. Click here to find out more.
Title
Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease
Published in
Molecular Neurodegeneration, December 2013
DOI 10.1186/1750-1326-8-45
Pubmed ID
Abstract

Mitochondrial impairment has been implicated in the pathogenesis of Huntington's disease (HD). However, how mutant huntingtin impairs mitochondrial function and thus contributes to HD has not been fully elucidated. In this study, we used striatal cells expressing wild type (STHdhQ7/Q7) or mutant (STHdhQ111/Q111) huntingtin protein, and cortical neurons expressing the exon 1 of the huntingtin protein with physiological or pathological polyglutamine domains, to examine the interrelationship among specific mitochondrial functions.

Twitter Demographics

The data shown below were collected from the profiles of 3 tweeters who shared this research output. Click here to find out more about how the information was compiled.

Mendeley readers

The data shown below were compiled from readership statistics for 100 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Portugal 1 1%
Chile 1 1%
Austria 1 1%
United Kingdom 1 1%
United States 1 1%
Unknown 95 95%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 22 22%
Researcher 16 16%
Student > Master 11 11%
Student > Bachelor 10 10%
Other 6 6%
Other 22 22%
Unknown 13 13%
Readers by discipline Count As %
Agricultural and Biological Sciences 28 28%
Biochemistry, Genetics and Molecular Biology 20 20%
Medicine and Dentistry 12 12%
Chemistry 6 6%
Neuroscience 5 5%
Other 12 12%
Unknown 17 17%

Attention Score in Context

This research output has an Altmetric Attention Score of 2. This is our high-level measure of the quality and quantity of online attention that it has received. This Attention Score, as well as the ranking and number of research outputs shown below, was calculated when the research output was last mentioned on 18 February 2014.
All research outputs
#13,204,453
of 21,162,624 outputs
Outputs from Molecular Neurodegeneration
#627
of 794 outputs
Outputs of similar age
#169,301
of 303,829 outputs
Outputs of similar age from Molecular Neurodegeneration
#15
of 35 outputs
Altmetric has tracked 21,162,624 research outputs across all sources so far. This one is in the 35th percentile – i.e., 35% of other outputs scored the same or lower than it.
So far Altmetric has tracked 794 research outputs from this source. They typically receive a lot more attention than average, with a mean Attention Score of 11.6. This one is in the 18th percentile – i.e., 18% of its peers scored the same or lower than it.
Older research outputs will score higher simply because they've had more time to accumulate mentions. To account for age we can compare this Altmetric Attention Score to the 303,829 tracked outputs that were published within six weeks on either side of this one in any source. This one is in the 41st percentile – i.e., 41% of its contemporaries scored the same or lower than it.
We're also able to compare this research output to 35 others from the same source and published within six weeks on either side of this one. This one has gotten more attention than average, scoring higher than 57% of its contemporaries.