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Inhalation of diesel exhaust does not exacerbate cardiac hypertrophy or heart failure in two mouse models of cardiac hypertrophy

Overview of attention for article published in Particle and Fibre Toxicology, October 2013
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Title
Inhalation of diesel exhaust does not exacerbate cardiac hypertrophy or heart failure in two mouse models of cardiac hypertrophy
Published in
Particle and Fibre Toxicology, October 2013
DOI 10.1186/1743-8977-10-49
Pubmed ID
Authors

Yonggang Liu, Wei-Ming Chien, Ivan O Medvedev, Chad S Weldy, Daniel L Luchtel, Michael E Rosenfeld, Michael T Chin

Abstract

Strong associations have been observed between exposure to fine ambient particulate matter (PM2.5) and adverse cardiovascular outcomes. In particular, exposure to traffic related PM2.5 has been associated with increases in left ventricular hypertrophy, a strong risk factor for cardiovascular mortality. As much of traffic related PM2.5 is derived from diesel exhaust (DE), we investigated the effects of chronic DE exposure on cardiac hypertrophy and heart failure in the adult mouse by exposing mice to DE combined with either of two mouse models of cardiac hypertrophy: angiotensin II infusion or pressure overload induced by transverse aortic banding.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 41 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Germany 1 2%
Unknown 40 98%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 9 22%
Student > Master 9 22%
Researcher 8 20%
Student > Bachelor 3 7%
Student > Doctoral Student 1 2%
Other 1 2%
Unknown 10 24%
Readers by discipline Count As %
Medicine and Dentistry 7 17%
Environmental Science 6 15%
Biochemistry, Genetics and Molecular Biology 3 7%
Agricultural and Biological Sciences 3 7%
Computer Science 2 5%
Other 5 12%
Unknown 15 37%