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X Demographics
Mendeley readers
Attention Score in Context
Title |
VPAC2 receptor agonist BAY 55-9837 increases SMN protein levels and moderates disease phenotype in severe spinal muscular atrophy mouse models
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Published in |
Orphanet Journal of Rare Diseases, January 2014
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DOI | 10.1186/1750-1172-9-4 |
Pubmed ID | |
Authors |
Jeremiah Hadwen, Duncan MacKenzie, Fahad Shamim, Kevin Mongeon, Martin Holcik, Alex MacKenzie, Faraz Farooq |
Abstract |
Spinal Muscular Atrophy (SMA) is one of the most common inherited causes of infant death and is caused by the loss of functional survival motor neuron (SMN) protein due to mutations or deletion in the SMN1 gene. One of the treatment strategies for SMA is to induce the expression of the protein from the homologous SMN2 gene, a rescuing paralog for SMA. |
X Demographics
The data shown below were collected from the profile of 1 X user who shared this research output. Click here to find out more about how the information was compiled.
Geographical breakdown
Country | Count | As % |
---|---|---|
Mexico | 1 | 100% |
Demographic breakdown
Type | Count | As % |
---|---|---|
Members of the public | 1 | 100% |
Mendeley readers
The data shown below were compiled from readership statistics for 29 Mendeley readers of this research output. Click here to see the associated Mendeley record.
Geographical breakdown
Country | Count | As % |
---|---|---|
United Kingdom | 1 | 3% |
United States | 1 | 3% |
Unknown | 27 | 93% |
Demographic breakdown
Readers by professional status | Count | As % |
---|---|---|
Student > Ph. D. Student | 8 | 28% |
Student > Master | 6 | 21% |
Researcher | 6 | 21% |
Other | 2 | 7% |
Professor | 1 | 3% |
Other | 2 | 7% |
Unknown | 4 | 14% |
Readers by discipline | Count | As % |
---|---|---|
Biochemistry, Genetics and Molecular Biology | 7 | 24% |
Medicine and Dentistry | 7 | 24% |
Agricultural and Biological Sciences | 5 | 17% |
Pharmacology, Toxicology and Pharmaceutical Science | 2 | 7% |
Neuroscience | 2 | 7% |
Other | 1 | 3% |
Unknown | 5 | 17% |
Attention Score in Context
This research output has an Altmetric Attention Score of 2. This is our high-level measure of the quality and quantity of online attention that it has received. This Attention Score, as well as the ranking and number of research outputs shown below, was calculated when the research output was last mentioned on 20 January 2014.
All research outputs
#14,187,012
of 22,739,983 outputs
Outputs from Orphanet Journal of Rare Diseases
#1,554
of 2,605 outputs
Outputs of similar age
#173,000
of 304,788 outputs
Outputs of similar age from Orphanet Journal of Rare Diseases
#42
of 58 outputs
Altmetric has tracked 22,739,983 research outputs across all sources so far. This one is in the 35th percentile – i.e., 35% of other outputs scored the same or lower than it.
So far Altmetric has tracked 2,605 research outputs from this source. They typically receive more attention than average, with a mean Attention Score of 7.5. This one is in the 36th percentile – i.e., 36% of its peers scored the same or lower than it.
Older research outputs will score higher simply because they've had more time to accumulate mentions. To account for age we can compare this Altmetric Attention Score to the 304,788 tracked outputs that were published within six weeks on either side of this one in any source. This one is in the 40th percentile – i.e., 40% of its contemporaries scored the same or lower than it.
We're also able to compare this research output to 58 others from the same source and published within six weeks on either side of this one. This one is in the 22nd percentile – i.e., 22% of its contemporaries scored the same or lower than it.