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Viable mouse gene ablations that robustly alter brain Aβ levels are rare

Overview of attention for article published in BMC Neuroscience, November 2010
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Title
Viable mouse gene ablations that robustly alter brain Aβ levels are rare
Published in
BMC Neuroscience, November 2010
DOI 10.1186/1471-2202-11-143
Pubmed ID
Authors

Jeremy H Toyn, Xu-Alan Lin, Mark W Thompson, Valerie Guss, Jere E Meredith, Sethu Sankaranarayanan, Nestor Barrezueta, John Corradi, Antara Majumdar, Daniel L Small, Melissa Hansard, Thomas Lanthorn, Ryan S Westphal, Charles F Albright

Abstract

Accumulation of amyloid-β (Aβ) peptide in the brain is thought to play a key pathological role in Alzheimer's disease. Many pharmacological targets have therefore been proposed based upon the biochemistry of Aβ, but not all are equally tractable for drug discovery.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 27 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 27 100%

Demographic breakdown

Readers by professional status Count As %
Researcher 8 30%
Professor > Associate Professor 5 19%
Student > Ph. D. Student 5 19%
Student > Master 3 11%
Student > Bachelor 2 7%
Other 4 15%
Readers by discipline Count As %
Agricultural and Biological Sciences 9 33%
Medicine and Dentistry 5 19%
Biochemistry, Genetics and Molecular Biology 4 15%
Neuroscience 4 15%
Nursing and Health Professions 1 4%
Other 2 7%
Unknown 2 7%