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Mendeley readers
| Title |
Expression of ATF3 and axonal outgrowth are impaired after delayed nerve repair
|
|---|---|
| Published in |
BMC Neuroscience, September 2008
|
| DOI | 10.1186/1471-2202-9-88 |
| Pubmed ID | |
| Authors |
Harukazu Saito, Lars B Dahlin |
| Abstract |
A delay in surgical nerve repair results in impaired nerve function in humans, but mechanisms behind the weakened nerve regeneration are not known. Activating transcription factor 3 (ATF3) increases the intrinsic growth state of injured neurons early after injury, but the role of long-term changes and their relation to axonal outgrowth after a delayed nerve repair are not well understood. ATF3 expression was examined by immunohistochemistry in motor and sensory neurons and in Schwann cells in rat sciatic nerve and related to axonal outgrowth after transection and delayed nerve repair (repair 0, 30, 90 or 180 days post-injury). Expression of the neuronal cell adhesion molecule (NCAM), which is expressed in non-myelinating Schwann cells, was also examined. |
Mendeley readers
The data shown below were compiled from readership statistics for 74 Mendeley readers of this research output. Click here to see the associated Mendeley record.
Geographical breakdown
| Country | Count | As % |
|---|---|---|
| United Kingdom | 1 | 1% |
| United States | 1 | 1% |
| Netherlands | 1 | 1% |
| Unknown | 71 | 96% |
Demographic breakdown
| Readers by professional status | Count | As % |
|---|---|---|
| Student > Ph. D. Student | 17 | 23% |
| Researcher | 14 | 19% |
| Student > Bachelor | 8 | 11% |
| Professor | 5 | 7% |
| Student > Master | 5 | 7% |
| Other | 14 | 19% |
| Unknown | 11 | 15% |
| Readers by discipline | Count | As % |
|---|---|---|
| Medicine and Dentistry | 21 | 28% |
| Agricultural and Biological Sciences | 20 | 27% |
| Neuroscience | 7 | 9% |
| Biochemistry, Genetics and Molecular Biology | 5 | 7% |
| Chemistry | 2 | 3% |
| Other | 6 | 8% |
| Unknown | 13 | 18% |