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The clinical course of alcoholic cirrhosis: effects of hepatic metabolic capacity, alcohol consumption, and hyponatremia – a historical cohort study

Overview of attention for article published in BMC Research Notes, September 2012
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Title
The clinical course of alcoholic cirrhosis: effects of hepatic metabolic capacity, alcohol consumption, and hyponatremia – a historical cohort study
Published in
BMC Research Notes, September 2012
DOI 10.1186/1756-0500-5-509
Pubmed ID
Authors

Peter Jepsen, Peter Ott, Per Kragh Andersen, Hendrik Vilstrup

Abstract

The cirrhosis complications hepatic encephalopathy, ascites, and variceal bleeding increase mortality but develop in random sequence. Therefore prognoses based on the presence or absence of these clinical complications are inherently inaccurate, and other determinants of the clinical course should be identified. Here we present our study of patho-etiological factors that may be causally involved in the development of specific complications to alcoholic cirrhosis; it was based on a model of cirrhosis pathophysiology encompassing hepatic metabolic capacity, continued alcohol consumption, and circulatory dysfunction.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 32 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Spain 1 3%
Denmark 1 3%
Australia 1 3%
Unknown 29 91%

Demographic breakdown

Readers by professional status Count As %
Student > Bachelor 5 16%
Student > Ph. D. Student 5 16%
Student > Postgraduate 5 16%
Researcher 3 9%
Student > Master 2 6%
Other 5 16%
Unknown 7 22%
Readers by discipline Count As %
Medicine and Dentistry 15 47%
Nursing and Health Professions 3 9%
Agricultural and Biological Sciences 2 6%
Pharmacology, Toxicology and Pharmaceutical Science 1 3%
Unspecified 1 3%
Other 2 6%
Unknown 8 25%